Filtros : "Ferraz, Jose G. P." Limpar

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  • Source: Antioxidants and Redox Signaling. Unidade: ICB

    Assunto: FARMACOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      WALLACE, John L. e FERRAZ, Jose G. P. e MUSCARÁ, Marcelo Nicolás. Hydrogen sulfide: an endogenous mediator of resolution of inflammation and injury. Antioxidants and Redox Signaling, v. 17, n. 1, p. 58-67, 2012Tradução . . Disponível em: https://doi.org/10.1089/ars.2011.4351. Acesso em: 02 jun. 2024.
    • APA

      Wallace, J. L., Ferraz, J. G. P., & Muscará, M. N. (2012). Hydrogen sulfide: an endogenous mediator of resolution of inflammation and injury. Antioxidants and Redox Signaling, 17( 1), 58-67. doi:10.1089/ars.2011.4351
    • NLM

      Wallace JL, Ferraz JGP, Muscará MN. Hydrogen sulfide: an endogenous mediator of resolution of inflammation and injury [Internet]. Antioxidants and Redox Signaling. 2012 ; 17( 1): 58-67.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1089/ars.2011.4351
    • Vancouver

      Wallace JL, Ferraz JGP, Muscará MN. Hydrogen sulfide: an endogenous mediator of resolution of inflammation and injury [Internet]. Antioxidants and Redox Signaling. 2012 ; 17( 1): 58-67.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1089/ars.2011.4351
  • Source: European Journal of Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      CAMARA, Paula R. S. et al. Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats. European Journal of Pharmacology, v. 589, n. 1-3, p. 245-250, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2008.05.004. Acesso em: 02 jun. 2024.
    • APA

      Camara, P. R. S., Ferraz, G. J. N., Franco-Penteado, C. F., Sbragia-Neto, L., Meirelles, L. R., Teixeira, S. A., et al. (2008). Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats. European Journal of Pharmacology, 589( 1-3), 245-250. doi:10.1016/j.ejphar.2008.05.004
    • NLM

      Camara PRS, Ferraz GJN, Franco-Penteado CF, Sbragia-Neto L, Meirelles LR, Teixeira SA, Muscará MN, Velloso LA, Antunes E, Ferraz JGP. Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats [Internet]. European Journal of Pharmacology. 2008 ; 589( 1-3): 245-250.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2008.05.004
    • Vancouver

      Camara PRS, Ferraz GJN, Franco-Penteado CF, Sbragia-Neto L, Meirelles LR, Teixeira SA, Muscará MN, Velloso LA, Antunes E, Ferraz JGP. Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats [Internet]. European Journal of Pharmacology. 2008 ; 589( 1-3): 245-250.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2008.05.004

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