Hepatic LC3 II/I ratio is not modulated in exercised mice (2020)
- Authors:
- USP affiliated authors: SILVA, ADELINO SANCHEZ RAMOS DA - EEFERP ; MARAFON, BRUNO BRIEDA - EEFERP ; PINTO, ANA PAULA - FMRP ; ROCHA, ALISSON LUIZ DA - FMRP ; ROVINA, RAFAEL LEMES - EEFERP
- Unidades: EEFERP; FMRP
- DOI: 10.33549/physiolres.934441
- Subjects: ESPORTES; TREINAMENTO FÍSICO; RESISTÊNCIA FÍSICA; FÍGADO
- Keywords: Autophagy; Concurrent training; Endurance; Liver; Resistance
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Physiological Research
- ISSN: 1802-9973
- Volume/Número/Paginação/Ano: v. 69, n. 6, p. 1103-1111, 2020
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by-nc
-
ABNT
MARAFON, Bruno Brieda et al. Hepatic LC3 II/I ratio is not modulated in exercised mice. Physiological Research, v. 69, n. 6, p. 1103-1111, 2020Tradução . . Disponível em: https://doi.org/10.33549/physiolres.934441. Acesso em: 23 maio 2024. -
APA
Marafon, B. B., Pinto, A. P., Rocha, A. L. da, Rovina, R. L., Pauli, J. R., Moura, L. P. de, et al. (2020). Hepatic LC3 II/I ratio is not modulated in exercised mice. Physiological Research, 69( 6), 1103-1111. doi:10.33549/physiolres.934441 -
NLM
Marafon BB, Pinto AP, Rocha AL da, Rovina RL, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Hepatic LC3 II/I ratio is not modulated in exercised mice [Internet]. Physiological Research. 2020 ; 69( 6): 1103-1111.[citado 2024 maio 23 ] Available from: https://doi.org/10.33549/physiolres.934441 -
Vancouver
Marafon BB, Pinto AP, Rocha AL da, Rovina RL, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Hepatic LC3 II/I ratio is not modulated in exercised mice [Internet]. Physiological Research. 2020 ; 69( 6): 1103-1111.[citado 2024 maio 23 ] Available from: https://doi.org/10.33549/physiolres.934441 - Impact of different physical exercises on the expression of autophagy markers in mice
- Moderate, but not excessive, training attenuates autophagy machinery in metabolic tissues
- Muscle endoplasmic reticulum stress in exercise
- Role of interleukin-6 in inhibiting hepatic autophagy markers in exercised mice
- Resposta do Nr1d1 em diferentes modelos de exercício físico agudo no músculo esquelético de camundongos
- Genetic deletion of IL‐6 increases CK‐MB, a classic cardiac damage marker, and decreases UPRmt genes after exhaustive exercise
- Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation
- Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts
- The combination of fasting, acute resistance exercise, and protein ingestion led to different responses of autophagy markers in gastrocnemius and liver samples
- Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage
Informações sobre o DOI: 10.33549/physiolres.934441 (Fonte: oaDOI API)
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