Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS (2018)
- Authors:
- USP affiliated authors: MARTINIS, BRUNO SPINOSA DE - FFCLRP ; TIRAPELLI, CARLOS RENATO - EERP
- Unidades: FFCLRP; EERP
- DOI: 10.1016/j.ejphar.2018.02.008
- Subjects: ESTRESSE OXIDATIVO; ETANOL; CITOCINAS; SEPSE; INTERLEUCINAS
- Keywords: Sub-lethal sepsis; Ethanol; iNOS; Oxidative stress
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: European Journal of Pharmacology
- ISSN: 0014-2999
- Volume/Número/Paginação/Ano: v. 825, p. 39-47, 2018
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
CERON, Carla S. et al. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, v. 825, p. 39-47, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2018.02.008. Acesso em: 23 maio 2024. -
APA
Ceron, C. S., Vale, G. T. do, Simplicio, J. A., Ricci, S. T., Martinis, B. S. de, Freitas, A. de, & Tirapelli, C. R. (2018). Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, 825, 39-47. doi:10.1016/j.ejphar.2018.02.008 -
NLM
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 maio 23 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008 -
Vancouver
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 maio 23 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008 - Vitamin C prevents the endothelial dysfunction induced by acute ethanol intake
- Nebivolol prevents vascular oxidative stress and hypertension in rats chronically treated with ethanol
- NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature
- Ethanol withdrawal induces anxiety-like effects: role of nitric oxide synthase in the dorsal raphe nucleus of rats
- Ethanol withdrawal increases blood pressure and vascular oxidative stress: a role for angiotensin type 1 receptors
- Chronic ethanol consumption induces erectile dysfunction: role of oxidative stress
- Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species
- Sweat an alternative matrix for amphetamines and methylenEdioxy derivatives analysis
- Determination of cocaine and metabolites in hair by column-switching LC-MS-MS analysis
- Investigation of Ayahuasca β-carboline alkaloids and tryptamine in sweat samples from religious community participants by GC-MS
Informações sobre o DOI: 10.1016/j.ejphar.2018.02.008 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas